Scientists have actually identified fructose as a central consider obesity, linking its function as a “survival switch” to weight gain and metabolic concerns. The research study shows fructose boosts food intake, damages mitochondria, and can result in diseases like diabetes and fatty liver illness
A recent study delves into the precise role fructose plays in developing obesity by taking a look at nature.
University of Colorado Anschutz Medical Campus researchers have actually officially identified a main channel to weight problems: fructose.
While fructoses contribution to obesity is well-known, a study recently released in Philosophical Transactions aggregates a large quantity of work to make a complete argument for how fructose drives obesity and illness such as diabetes and fatty liver illness.
” This is a thorough review on a hypothesis that puts nature at the center of weight gain, taking a look at how fructose works in a different way than other nutrients by lowering active energy,” says Richard Johnson, MD, professor at the University of Colorado School of Medicine and research study lead author. “We identify a recently discovered function of fructose in survival that stores fuel in case resources become scarce. This is called the survival switch,” he says.
Fructose is the source of sweet taste in fruit however is primarily consumed in Western society as table sugar and high fructose corn syrup, much different than the nutrition consumed by our forefathers ahead of lean winter months. Johnson and scientists posited that fructose works in a different way than other nutrients by lowering active energy, and damaging mitochondria.
Study results reveal that fructose stimulates food intake and lowers resting basal metabolism, much like an animal preparing to hibernate. Further, results show that the administration of fructose can lead to weight gain, insulin resistance, raised high blood pressure, and fatty liver amongst a host of other metabolic-related issues.
” This work assembles in one place the full argument for how a specific carbohydrate, fructose, might have a main role in driving weight problems and diabetes,” says Johnson. “This is a very exciting, new hypothesis that unifies other hypotheses to indicate the specific function fructose plays in the beginning of obesity. And we can trace it back to our ancestors, along with gain from hibernating animals, precisely how fructose triggers this switch within us.”
Recommendation: “The fructose survival hypothesis for weight problems” by Richard J. Johnson, Miguel A. Lanaspa, L. Gabriela Sanchez-Lozada, Dean Tolan, Takahiko Nakagawa, Takuji Ishimoto, Ana Andres-Hernando, Bernardo Rodriguez-Iturbe and Peter Stenvinkel, 24 July 2023, Philosophical Transactions of the Royal Society B Biological Sciences.DOI: 10.1098/ rstb.2022.0230.
” This is a thorough review on a hypothesis that puts nature at the center of weight gain, examining how fructose works differently than other nutrients by decreasing active energy,” states Richard Johnson, MD, teacher at the University of Colorado School of Medicine and study lead author.” This work puts together in one location the complete argument for how a specific carb, fructose, might have a central role in driving obesity and diabetes,” says Johnson. “This is an extremely amazing, brand-new hypothesis that joins other hypotheses to point to the specific function fructose plays in the onset of weight problems.
